08/09/2021
Joint or navel ill
Joint ill occurs in lambs up to one month of age. Affected lambs are often lame in several joints, usually limb joints, including fetlocks, knees, hocks, and stifles. Affected joints are hot and painful. The lambs are dull, feverish, and clearly unthrifty. Some may have swollen, infected navels, while others may have symptoms of pneumonia or meningitis.
The infection is usually caused by strains of streptococci, though coliforms and occasionally Actinomyces pyogenes may be isolated. Affected lambs should be treated with a long-acting penicillin. Joint ill is prevented by good hygiene and using a navel dip, such as betadine or gentle iodine.
Lameness
It has been estimated that 80 percent of the flocks in Great Britain have lame sheep. Lameness can be a sign of several foot conditions – some of which are very serious – as well as some other problems. These include foot rot and scald, strawberry foot, foot abscess, foot-and-mouth disease, bluetongue, ovine interdigital dermatitis (looks like scald), sore mouth, arthritis, nutritional deficiencies, mineral excesses, and physical injuries. The more common foot problems can be avoided or minimized if good husbandry practices are followed. Regular hoof inspection and foot paring will prevent many problems.
Laminitis (founder)
Lameness related to laminitis is caused by an inadequate flow of blood in the foot. Signs are heat in the feet. Laminitis is normally associated with digestive problems resulting from excessive intake of grain (grain overload/acidosis), which usually masks the effects on the feet. Such animals usually die before the feet become involved. Recovered animals may exhibit foot growth and/or permanent lameness. Feeding management is key to the prevention of laminitis/founder.
Listeriosis (circling disease)
Listeria monocytogenes, the bacteria that causes listeriosis, is widely distributed in nature and is found in soil, feedstuffs, and f***s from healthy animals. Listeriosis is most commonly associated with the feeding of moldy silage or spoiled hay, but because the organism lives naturally in the environment, listeriosis may occur sporadically.
Listeriosis usually presents itself as encephalitis (inflammation of the brain) but may also cause abortion in ewes. Sheep with the neurological form of the disease become depressed and disoriented. They may walk in circles with a head tilt and facial paralysis. Mortality is high and treatment (high doses of antibiotics) is generally not effective.
Mastitis (hard bag, blue bag)
Mastitis is an inflammation (or infection) of the mammary gland (udder) which is usually caused by a bacterial infection. The bacteria that most commonly cause mastitis in ewes are Staphylococcus aureus and Pasteurella hemolytica. There are two types of mastitis: acute and chronic. The glands of ewes with acute mastitis may be discolored and dark, swollen and very warm. The affected ewe may be reluctant to walk, may hold up one rear foot, and may not permit her lambs to nurse. Ewes with chronic mastitis often go undetected.
While no drugs are approved for sheep, mastitis is usually treated with intramammary infusions of antibiotics, systemic antibiotics, and anti-inflammatory drugs. There is no vaccine for mastitis. It is best prevented by good management and sanitation. Heavy milkers are more prone to mastitis. There is also a genetic component.
Measles
(sheep measles, cysticerosis)
Sheep measles (Cysticercus ovis) is the intermediate or larval stage of the cestode (tapeworm) Taenia ovis, the adult stage of which is found in the small intestine of dogs (sheep host the larvae stage). Sheep measle lesions are found in the heart, diaphragm and other muscles of sheep and goats. Although not considered to be a human health hazard, carcasses can be condemned on account of sheep measles.
There are no clinical signs of cysticerosis in sheep. Currently diagnosis is only made by finding the cysts at slaughter. To prevent sheep measles, dogs and other canines should not be allowed to feed on sheep or goat carcasses. Dogs should be dewormed for tapeworms. Any dog given access to the farm should be required to be dewormed.
Milk fever (hypocalcemia, parturient paresis)
Milk fever is a metabolic disease affecting mostly pregnant ewes near term when calcium requirements are the highest. It is most commonly caused by an inadequate intake of calcium, but can also be caused by a ewe's inability to mobilize calcium reserves prior to or after lambing. Milk fever presents similar symptoms as pregnancy toxemia but can be differentiated by the affected ewe's response to calcium therapy.
Ewes in the early stages of milk fever can be administered calcium gluconate subcutaneously. More seriously affected ewes will require intravenous calcium and other supportive therapies. Milk fever can be prevented by providing proper levels of calcium in ewe diets, especially during late gestation.
Ovine progressive pneumonia (OPP, lunger disease. Maedi-Visna)
Ovine progressive pneumonia is a slow developing viral disease that is characterized by progressive weight loss, difficulty in breathing and development of lameness, paralysis, and hard bag. It is very closely related to caprine arthritis-encephalitis virus (CAE) and is caused by a retrovirus. The OPP virus closely resembles Maedi-Visna which is a similar slow or retrovirus found in other parts of the world.
OPP is transmitted laterally to other susceptible animals or to offspring through ingestion of infected milk and colostrum. Veterinary diagnostic laboratory assistance is required for diagnosis. There is no treatment, but OPP can be eliminated from the herd using annual blood testing and removal of positive animals and removal of the lambs from the ewes prior to suckling.
It is estimated that over 50% of the flocks in the U.S. are infected with OPP with the number of sheep infected within a positive flock anywhere between 1% to 70%. However, the vast majority of infected sheep will never show respiratory disease or a wasting syndrome.
Pink eye (infectious keratoconjunctivitis)
Pinkeye is a highly contagious disease affecting the eyes of sheep. Pinkeye may result from many different infective agents: Chlamydia, certain viruses, and mycoplasma. The disease will usually complete its course in three weeks in individual sheep. The use of eye medications containing antibiotics may be helpful in individual cases. There are no effective vaccines available, as the agent that causes pinkeye in sheep and goats is different from the one that causes it in cattle.
Read Infectious Keratoconjunctivitis (Pinkeye) =>
Pizzle Rot (sheath rot)
Pizzle rot is an infection in the sheath area of the ram. It is caused by the bacteria, Corynebacterium renale or one from that group. The other factor is high protein diets (>16 percent). Ammonia produced by the excess urea in the ram's urine can cause severe irritation and ulceration of the skin around the preputial opening. The debris from the ulcer form a crust which may block the opening to the prepuce. Pizzle rot can affect a ram's desire and ability to mate.
Plant poisoning
It is important to consider plant toxicities when diagnosing death losses. Many plants are toxic or potentially toxic to sheep. Some plants accumulate toxins during specific times of their growing cycle or after periods of environmental stress. The incidence of plant poisoning in livestock tends to increase when normal forages are scarce, causing animals to eat plants that they would not normally eat.
The signs of plant poisoning are as varied as the plants themselves and may mimic other diseases. Many poisonous plants cause sudden death. Some plants cause photosensitization (a severe skin reaction). Other poisonous plants affect the nervous system. Some plant poisonings can be treated if signs are recognized early. For many plant toxins, there are no treatments.
Pneumonia
(respiratory disease complex, pasteurellosis, shipping fever)
Pneumonia is second in importance to diseases of the digestive tract. Pneumonia is a respiratory complex with no single agent being solely responsible for the disease. The most common bacteria isolated from respiratory infections is Pasteurella haemolytica or Pasteurella multocida or both. Affected animals become depressed and go off feed. They may cough and show some respiratory distress. Temperatures are usually over 104°F. The disease may be acute with sudden deaths or take a course of several days. Pneumonia is treated with antibiotics. There is a vaccine for Pasteurella.
Polioencephalomalacia (PEM, CCN, polio, cerebrocortical necrosis)
Polioencephalomalacia is a disease of the central nervous system, caused by a vitamin B1 (thiamine) deficiency. Since the rumen manufactures B vitamins, polio is not caused by insufficient thiamine, but rather the inability to utilize it. The most common symptom of polio is blindness and star-gazing.
Polio most commonly occurs in lambs that are consuming high concentrate diets. Polio can also occur in sheep that consume plants that contain a thiamase inhibitor. Excessive use of amprolium (Corid) can cause polio. Polio symptoms mimic other neurological disease conditions, but a differential diagnosis can be made based on the animals' response to injections of vitamin B1.
Polyarthritis
Polyarthritis is an infectious disease of nursing lambs, recently weaned lambs, and feedlot lambs. Symptoms are stiffness, reluctance to move, depression, loss of body weight, and conjunctivitis. Clinically the disease is primarily characterized by stiffness and by conjunctivitis. Body temperatures over 104°F are common. Lambs can be treated with several different broad-spectrum antibiotics or tetracycline drugs.
Pregnancy Toxemia
(ketosis, twin lamb disease, lambing paralysis, hypoglycemia)
Pregnancy toxemia is a metabolic disease that affects ewes during late gestation. It most commonly affects ewes, overfat ewes, older ewes, and/or ewes carrying multiple fetuses. It is caused by an inadequate intake of energy during late pregnancy, when the majority of fetal growth is occurring.
Treatment is to increase the blood sugar supply to the body by administering glucose intravenously or propylene glycol or molasses orally. In extreme cases, removal of the fetuses is the only recourse to save the ewe and lambs.
Pregnancy toxemia can be prevented by providing adequate energy to ewes during late gestation, usually ½ to 1 lb. of grain per head per day, more for high producing ewes. Adequate feeder space is also necessary to ensure all ewes are able to consume enough feed.
Read Pregnancy Toxemia in Ewes and Does =>
Rabies
Rabies is a viral disease of the central nervous system of mammals, spread by contact with saliva from an infected animal, usually through bites or scratches, abrasions, or open wounds in the skin. Domestic animals may become exposed during normal grazing or roaming. Sheep have symptoms similar to cattle, and sometimes vigorously pull their wool. Livestock and horse owners may decide to vaccinate their animals if they are often exposed to potentially rabid wild or domestic animals.
Generally, production animals, such as dairy cow herds and sheep flocks, are not vaccinated because the potential risks are usually lower than the annual costs of vaccination and because human contact with individual animals is low. Small groups of valuable purebred animals may be an exception. Producers who lease their animals for grazing or use their animals for exhibition should consider vaccinating. In recent years, a few states have required vaccination for rabies before an animal (including some livestock) can be exhibited publicly.
Re**al Prolapse
A re**al prolapse is protrusion of the re**al tissue through the exterior of the body. It usually begins as a small round area that sticks out when the lamb lays down or coughs. In extreme cases, the intestines can pass through the opening and the disease can be fatal. There are many predisposing factors to re**al prolapses, including genetics, short tail docks, coughing, weather, stress, and high concentrate diets.
Re**al prolapses tend to occur more in ewe lambs than wether lambs and more in black-faced sheep than white-faced sheep. It is a heritable trait, about 10 percent. Lambs on high concentrate diets are more prone. In fact, a link between ultra-short tail docking and concentrate feeding has been scientifically established. Usually, lambs with pr*****ed re**ums are prematurely slaughtered or sent to market. It is possible to repair a re**al prolapse by amputating the pr*****ed part of the re**um. These lambs should not be kept for breeding.
Read Re**al prolapses: a complex problem with many contributing factors
Ringwomb
Ringwomb is when the cervix fails to dilate sufficiently to allow delivery of the lamb(s). While sometimes the cervix of affected ewes can be opened with gentle pressure or the injection of hormones, usually such efforts prove futile and a caesarian section to remove the lambs is the only option that will produce a successful outcome for both ewes and lambs. Unfortunately, little is known about the cause of ringworm and how to prevent it. There is some evidence to suggest that ringwomb has a genetic component.
Ringworm (club lamb fungus, wool rot, and lumpy wool)
Club lamb fungus is a highly contagious fungal infection of the skin of sheep. It is primarily a problem with show lambs that are frequently slick sheared. Club Lamb Fungus is caused by fungus of the genus Trichophyton. Infection occurs when the fungus invades the skin and hair (wool) follicles. Fungal spores are transmitted by contaminated clippers, blankets, combs, bedding, bunks, and pens. Lesions can appear anywhere, however, most are found on the head, neck, and back. The infection is susceptible to anti-fungal agents. Club lamb fungus causes a nasty ringworm infection in people.
Ryegrass staggers
Ryegrass staggers is a disease of grazing animals that causes muscle spasms, loss of muscle control and paralysis. It is caused by a group of toxins that accumulates in the leaf sheaths of perennial ryegrass. The toxins are produced by a native fungus called ryegrass endophyte, Neotyphodium lolii, that grows within the leaves, stems and seeds of perennial ryegrass. Sheep and cattle are most commonly affected but horses, alpaca. and deer are also susceptible.
Ryegrass staggers has not been recorded in goats. Affected animals have a stiff gait or are unable to walk. They may injure or kill themselves in transit. The toxins can induce high body temperatures thus animals will try to cool themselves. Younger animals tend to be worst affected. The symptoms of ryegrass staggers usually develop 7-14 days after livestock stock start grazing the toxic parts of the plant. Prolonged exposure to toxic pasture can lead to permanent neurological damage.
Scrapie
Scrapie is a degenerative, fatal disease affecting the central nervous system of sheep (and goats). The causative agent is believed to be a prion, a misshapen protein. The disease is spread via placenta, from the dam to her offspring and other lambs (and kids) that come into contact with her birthing fluids, placenta, and bedding soiled with birthing fluids. There is no treatment for scrapie. Affected animals always die.
While the occurrence of scrapie in the U.S. sheep flock is low and getting lower all the time, it is a disease of regulatory concern. This is because scrapie is a member of a family of diseases called "transmissible spongiform encephalopathies (TGE's), which also includes chronic wasting disease (in mule deer and elk), mad cow disease (bovine spongiform encephalopathy) and classic and new variant Creutzfeldt-Jacob's Disease (in humans).
Producers of breeding stock are encouraged to enrolled in the voluntary scrapie flock certification program, which after five years of scrapie-free monitoring, enables a flock to be certified "scrapie-free." Furthermore, while scrapie is not a genetic disease, a sheep's genetic make-up influences its susceptibility to scrapie if exposed to the infective agent. Therefore, sheep can be tested for scrapie resistance.
Scrotal hernia
A scrotal hernia is when the ram's intestines slip through the inguinal rings into the sc***um. The condition causes an enlargement of the sc***um. Scrotal hernias may be congenital or acquired. They are thought to be caused by trauma. While it may be possible to surgically repair a scrotal hernia, a more practical option would be harvest them for meat. Since heredity probably plays a role in the occurrence of a scrotal hernia, it is probably prudent to cull rams that sire lambs that develop hernias.
Septic pedal arthritis
Septic pedal arthritis is a bacterial infection that usually gains entry to the distal interphalangeal (pedal) joint from an interdigital legion which then tracks across the joint to discharge above the coronary band. The foot is swollen with obvious widening of the interdigital space and a discharging sinus(es) above the coronary band. In chronic cases, there is considerable widening of the interdigital space and loss of hair around the coronary band. Chronic cases usually do not respond to antibiotic therapy. Digit amputation by a veterinarian is usually necessary.
Soremouth
(contagious ecthyma, scabby mouth, pustular dermatitis, orf)
Soremouth is the most common skin disease affecting sheep (and goats). It is a highly contagious viral infection that can also produce painful lesions in people. The virus causes scab formation on the skin, usually around the mouth, nostrils, eyes, mammary gland, and v***a. It first appears as tiny red nodules, usually at the junction of the lips. Treatment is usually unrewarding, though WD-40 has been advocated as a treatment. The disease will usually run its course in 1 to 4 weeks.
Effective vaccines are available. The vaccine is applied to a woolless area in the inside of the ear or under a leg where it cannot spread to the mouths of other animals. Once the vaccine is used on the premises, it should be continued yearly. Flocks that have not experienced soremouth should not vaccinate for soremouth since the vaccine introduces the virus to the farm.
Spider Syndrome
(spider lamb disease, ovine hereditary chondrodysplasia)
Spider lamb syndrome is a genetic condition that causes lambs to have severe malformations of the skeletal system. These animals have very fine bone, crooked legs and a crooked spinal column, and a distinct lack of muscular development. They usually do not survive to full maturity.
The cause of the condition appears to be genetic alteration due to selection for extreme length and height in show sheep. The disease is found predominantly in black-faced lambs: 75% Suffolk and 25% Hampshire. In order to have this disease, lambs must inherit a recessive gene from each parent. Several labs offer genetic testing for spider lamb disease.
Urinary calculi (water belly, urolithiasis, calculosis)
Urinary calculi is a metabolic disease of wethers and rams characterized by the formation of calculi (stones) within the urinary tract. Blockage of the urethra by calculi causes retention of urine, abdominal pain, distention and rupture of the urethra or bladder. Left untreated, it can cause death.
The most common cause of urinary calculi is feeding rations with high phosphorus levels. Grain and oilseeds are usually high in phosphorus and low in calcium, whereas forages, especially legumes, have a much more desirable ratio. The ratio of calcium to phosphorus in the ration should be at least 2:1. Providing the proper balance of minerals in the ration is preferred to offering minerals free choice, since there is no guarantee animals will consume adequate amounts of free choice mineral.
Sheep rations should always include roughage, ideally long-stemmed forage. The addition of ammonium chloride (a urine acidifier) to the ration will aid in preventing urinary calculi. It is also important that animals have an ample supply of clean, potable water. The addition of salt to the ration will increase water intake and decrease stone formation.
Ram lambs that are castrated at an early age are at increased risk for developing urinary calculi, as their urethras do not develop as fully. However, for animal welfare reasons, late castration is not advocated, as almost all cases of urinary calculi can be prevented with proper nutrition.
Read Urinary Calculi in Sheep and Goats =>
Uterine prolapse
A uterine prolapse is when the womb (uterus) is turned inside out and pushed through the birth canal by abdominal strainings of the ewe. It may occur immediately after lambing or several days later. A uterine prolapse is life-threatening. Before the pr*****ed uterus can be put back into the ewe, the ewe's hindquarters should be raised. The uterus should be cleaned with a warm, soapy, disinfectant solution prior to replacement and should be replaced before the tissues become dry or chilled. Pouring water into the uterus will help to ensure that the tips of the horns are unfolded. Affected ewes should be given antibiotics and oxytocin. Unlike ewes that prolapse their va**nas, it is okay to keep a ewe that has pr*****ed her uterus.
Vaginal Prolapse
Vaginal prolapses (protrusion of the va**na) are most commonly observed during the last month of pregnancy or shortly after lambing. Many factors have been implicated in the cause of va**nal prolapse, such as hormonal/metabolic imbalances, overfat/overthin body condition, bulky feeds, lack of exercise, dystocia in previous pregnancies, increased abdominal pressure and fetal burden. Prolapses often recur in subsequent pregnancies.
The exposed va**na of affected ewes should be washed with soapy disinfectant solution and forced back into the ewe. A bearing retainer or "spoon" can be inserted and secured in the ewe to prevent further prolapsing. There are harnesses that can be put on ewes to prevent further prolapses. Sutures are another option. Sutures must be removed in order for the ewe to lamb. The ewes can lamb with the spoon or harness in place, but it is better to remove them. Affected ewes and their offspring should probably not be kept in the flock for breeding animals due to the hereditary nature of the problem.
White muscle disease
(WMD, nutritional muscular dystrophy, nutritional myopathy, stiff lamb disease)
White muscle disease is a degeneration of the skeletal and cardiac muscles of lambs. It is caused by a deficiency of selenium, vitamin E, or both and can be a problem wherever selenium levels in the soil are low or the diet is deficient in selenium. Symptoms are stiffness of the hind legs with an arched back and tucked in flanks. Treatment is the administration of selenium and vitamin E by injection.
Feed rations should be evaluated to determine if they are providing adequate levels of selenium and vitamin E. If dietary levels of selenium are inadequate, lambs can be given an injection of selenium and vitamin E shortly at birth. Dietary supplementation of selenium is usually preferred to selenium injections. There is a narrow margin between selenium deficiency and selenium toxicity.